Skip to main content
ARS Home » Midwest Area » East Lansing, Michigan » Sugarbeet and Bean Research » Research » Publications at this Location » Publication #127354


item Yu, Yi
item Mcgrath, J Mitchell - Mitch

Submitted to: Plant and Animal Genome Conference Proceedings
Publication Type: Abstract Only
Publication Acceptance Date: 11/8/2001
Publication Date: N/A
Citation: N/A

Interpretive Summary:

Technical Abstract: Seedling diseases contribute to significant stand losses in sugar beet. Aphanomyces cochlioides causes one seedling disease, and genetic tolerance has been deployed but not characterized. We developed a segregating population of 145 F2 individuals from a cross between sugar beet and a resistant wild beet accession, and followed the segregation of Aphanomyces reaction, as determined by a progeny test from 51 F2 individuals where see was available, with each of the 192 AFLP polymorphisms scored in F2 plants. 178 AFLP markers were linked on 9 groups covering 712 cM total. QTL analyses indicate a single significant region for Aphanomyces reaction using a novel assay. Additional traits such as germination speed and pigment intensification also showed significant QTLs. This information will be used to refine inheritance models for seedling disease resistance in breeding enhanced germplasm.