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ARS Home » Southeast Area » Gainesville, Florida » Center for Medical, Agricultural and Veterinary Entomology » Chemistry Research » People & Locations » Charles Hunter

Charles T Hunter
Chemistry Research
Research Geneticist

Phone: (352) 374-5858
Fax: (352) 374-5707

1700 S.W. 23RD DR
GAINESVILLE , FL 32608
Projects
Molecular and Biochemical Characterization of Biotic and Abiotic Stress on Plant Defense Responses in Maize
In-House Appropriated (D)
  Accession Number: 434496

Publications (Clicking on the reprint icon Reprint Icon will take you to the publication reprint.)
Pesticides on the inside: Exploiting the natural chemical defenses of maize against insect and microbial pests -
The W22 genome: a foundation for maize functional genomics and transposon biology -
Contrasting insect attraction and herbivore-induced plant volatile production in maize -
Block, A.K., Hunter Iii, C.T., Rering, C.C., Christensen, S.A., Meagher Jr, R.L. 2018. Contrasting insect attraction and herbivore-induced plant volatile production in maize. Planta. https://doi.org/10.1007/s00425-018-2886-x.
Commercial hybrids and mutant genotypes reveal complex protective roles for inducible terpenoid defenses -
Christensen, S.A., Sims, J., Vaughan, M.M., Hunter Iii, C.T., Block, A.K., Willett, D.S., Alborn, H.T., Huffaker, A., Schmelz, E.A. 2018. Commercial hybrids and mutant genotypes reveal complex protective roles for inducible terpenoid defenses. Journal of Experimental Botany. doi:10.1093/jxb/erx495.
Maize w3 disrupts homogentisate solanesyl transferase (ZmHst) and reveals a plastoquinone-9 independent path for phytoene desaturation and tocopherol accumulation in kernels -
Hunter Iii, C.T., Saunders, J., Magallanes-Lundback, M., Christensen, S.A., Willett, D.S., Stinard, P.S., Li, Q., Lee, K., Dellapenna, D., Koch, K.E. 2018. Maize w3 disrupts homogentisate solanesyl transferase (ZmHst) and reveals a plastoquinone-9 independent path for phytoene desaturation and tocopherol accumulation in kernels. Plant Journal. doi:10.1111/tpj.13821.
Herbivore derived fatty acid-amides elicit reactive oxygen species burst in plants -
Block, A.K., Christensen, S.A., Hunter Iii, C.T., Alborn, H.T. 2017. Herbivore derived fatty acid-amides elicit reactive oxygen species burst in plants. Journal of Experimental Botany. doi:10.1093/jxb/erx449.
Fungal and herbivore elicitation of a newly identified maize sesquiterpenoid, zealexin A4, is constrained by abiotic stress -
Christensen, S.A., Huffaker, A., Sims, J., Hunter Iii, C.T., Block, A.K., Vaughan, M.M., Willett, D.S., Mylroie, E., Williams, P.C., Schmelz, E.A. 2017. Fungal and herbivore elicitation of a newly identified maize sesquiterpenoid, zealexin A4, is constrained by abiotic stress. Planta. doi:10.1007/s00425-017-2830-5.
Seteria viridis as a model for pathogen resistance in the Poaceae -
Investigating the roles of jasmonic acid and cytokinin in maize leaf growth control -
Dissecting a new connection between cytokinin and jasmonic acid in control of leaf growth -
Laboratory techniques in plant molecular biology taught with UniformMu insertion alleles of maize -
Transposon elements in maize white seedling 3, allele w3-8686, w3-kermicle-1 and w3-88-89-3563-33 -
Homogentisate solanesyl transferase (HST) cDNA’s in maize -
Maize white seedling 3 results from disruption of homogentisate solanesyl transferase -
Maize pathogens suppress inducible phytoalexin production to thwart innate plant immunity -
A maize death acid, 10-oxo-11-phytoenoic acid, is the predominant cyclopentenone signal present during multiple stress and developmental conditions -
Christensen, S.A., Huffaker, A., Hunter III, C.T., Alborn, H.T., Schmelz, E. 2016. A maize death acid, 10-oxo-11-phytoenoic acid, is the predominant cyclopentenone signal present during multiple stress and developmental conditions. Plant Signaling and Behavior. 11(2):e1120395.
Over 10,000 new maize mutants added to the uniformMu public resource: now 67,000 total Mu insertions with 42% genome coverage -
A zebra-band phenotype in maize can be suppressed in constant light, and results from mutation of a PPOXlike gene (protophorphyrinogen oxidase IX-like) for porphyrin biosynthesis -