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ARS Home » Midwest Area » Madison, Wisconsin » Vegetable Crops Research » Research » Publications at this Location » Publication #303843
Title: Using comparative genomics to develop a new model of understanding Verticillium wilt resistance in potato

Author
item MEIER, AUSTIN - University Of Wisconsin
item Halterman, Dennis

Submitted to: American Phytopathological Society Abstracts
Publication Type: Abstract Only
Publication Acceptance Date: 3/17/2014
Publication Date: 8/9/2014
Citation: Meier, A., Halterman, D.A. 2014. Using comparative genomics to develop a new model of understanding Verticillium wilt resistance in potato [abstract]. American Phytopathological Society Abstracts. Paper No. 80-O.

Interpretive Summary:

Technical Abstract: Verticillium wilt (VW) is a persistent and costly disease affecting potato production. VW is caused by the soilborne fungi Verticillium dahliae and V. albo-atrum. Because of the difficulty in dealing with this disease, host resistance offers a low input, effective method of control. Most potato cultivars grown in the US are very susceptible, but sources of resistance have been identified in several wild relatives. In tomato, resistance to VW is conferred by Ve1, a member of a gene family encoding two membrane-bound, extracellular receptors. Homologs of Ve have been identified in wild and cultivated potato, including DM1-3, from the S. tuberosum group Phureja and reference for the potato genome sequence. Comparison of the Ve locus on chromosome 9 of VW susceptible DM1-3 and the resistant diploid hybrid C287 identified structural variation that could determine Ve gene function. Using comparative genomics and gene-expression analyses, we have developed a model for VW resistance and susceptibility in potato.