Chang, P-K., Hua, S.T., Sarreal, S.L., Scharfenstein, L.L., Li, R.W. 2015. The effect of 2-phenylethanol treatment on Aspergillus flavus transcriptome. National Center for Biotechnology Information (NCBI). Accession: SRP056528.
Chang, P.-K., Scharfenstein, L.L., Solorzano, C.D., Abbas, H.K., Hua, S.S., Jones, W.A., Zablotowicz, R.M. 2015. High sequence variations in the region containing genes encoding a cellular morphogenesis protein and the repressor of sexual development help to reveal origins of Aspergillus oryzae. International Journal of Food Microbiology. 200:66-71.
Zhang, H., Scharfenstein, L.L., Carter Wientjes, C.H., Chang, P., Zhang, D., Meng, X., Yu, J. 2015. Lack of aflatoxin production by Aspergillus flavus on a resistant peanut line is associated with delayed expression of aflatoxin genes. World Mycotoxin Journal. 8(3):335-340.
Chang, P.-K., Scharfenstein, L.L., Mack, B.M., Yu, J., Ehrlich, K. 2014. Transcriptomic profiles of Aspergillus flavus CA42, a strain that produces small sclerotia, by decanal treatment and after recovery. Fungal Genetics and Biology. 68:39-47.
Chang, P-K., Scharfenstein, L.L., Yu, J., Mack, B.M., Ehrlich, K.C., Losada, L., Nierman, W.C., Bhatnagar, D. 2014. Transcriptomic profiling of decanal effects on Aspergillus flavus gene expression in development and secondary metabolism. National Center for Biotechnology Information (NCBI). Accession: SRX471362.
Chang, P-K., Scharfenstein, L.L., Li, P., Ehrlich, K. 2013. Aspergillus flavus VelB acts distinctly from VeA in conidiation and may coordinate with FluG to modulate sclerotial production. Fungal Genetics and Biology. 58-59:71-79.
Chang, P-K., Scharfenstein, L.L., Mack, B.M., Ehrlich, K. 2012. Deletion of the Aspergillus flavus orthologue of A. nidulans fluG reduces conidiation and promotes production of sclerotia but does not abolish aflatoxin biosynthesis. Applied and Environmental Microbiology. 78(21):7557-7563.
Chang, P., Abbas, H.K., Weaver, M.A., Ehrlich, K., Scharfenstein, L.L., Cotty, P.J. 2012. Identification of genetic defects in the atoxigenic biocontrol strain Aspergillus flavus K49 reveals the presence of a competitive recombinant group in field populations. International Journal of Food Microbiology. 154:192-196.
Chang, P.-K., Scharfenstein, L.L., Ehrlich, K., Wei, Q., Bhatnagar, D., Ingber, B.F. 2012. Effects of laeA deletion on Aspergillus flavus conidial development and hydrophobicity may contribute to loss of aflatoxin production. Fungal Biology. 116:298-307.
Chang, P.-K., Scharfenstein, L.L., Luo, M., Mahoney, N.E., Molyneux, R.J., Yu, J., Brown, R.L., Campbell, B.C. 2010. Loss of msnA, a putative stress regulatory gene, in Aspergillus parasiticus and Aspergillus flavus increased production of conidia, aflatoxins and kojic acid. Toxins. 3:82-104.
Chang, P., Scharfenstein, L.L., Wei, Q., Bhatnagar, D. 2010. Development and Refinement of a High-Efficiency Gene-Targeting System for Aspergillus flavus. Journal of Microbiological Methods. 81:240-246.